- 產(chǎn)品描述
弓形蟲IgG免疫熒光試劑盒(檢測貓)
Toxoplasma IFA Feline IgG Kit
廣州健侖生物科技有限公司
主要用途:用于檢測貓血清中的弓形蟲IgG抗體
產(chǎn)品規(guī)格:12 孔/張,10 張/盒
主要產(chǎn)品包括:包柔氏螺旋體菌、布魯氏菌、貝納特氏立克次體、土倫桿菌、鉤端螺旋體、新型立克次體、恙蟲病、立克次體、果氏巴貝西蟲、馬焦蟲、牛焦蟲、利什曼蟲、新包蟲、弓形蟲、貓流感病毒、貓冠狀病毒、貓皰疹病毒、犬瘟病毒、犬細小病毒等病原微生物的 IFA、MIF、ELISA試劑。
弓形蟲IgG免疫熒光試劑盒(檢測貓)
我司還提供其它進口或國產(chǎn)試劑盒:登革熱、瘧疾、西尼羅河、立克次體、無形體、蜱蟲、恙蟲、利什曼原蟲、RK39、漢坦病毒、深林腦炎、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。
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JL-FL38 | parkeri立克次體IgG ELISA | R. parkeri IgG ELISA Kit |
JL-FL39 | montanensis立克次體IgG ELISA | R. montanensis IgG ELISA Kit |
JL-FL40 | EB病毒衣殼IgG免疫熒光玻片試劑盒 | EBV Viral Capsid IgG IFA Kit |
JL-FL41 | EB病毒衣殼IgM免疫熒光玻片試劑盒 | EBV Viral Capsid IgM IFA Kit |
JL-FL42 | EB病毒早期抗原IgG免疫熒光玻片試劑盒 | EBV Early Antigens IgG IFA Kit |
JL-FL43 | 鉤端螺旋體IgG免疫熒光試劑盒 | Leptospira IgG IFA Kit |
JL-FL44 | 鉤端螺旋體IgM免疫熒光試劑盒 | Leptospira IgM IFA Kit |
JL-FL45 | 果氏巴貝西蟲免疫熒光玻片 | Babesia microti IFA Substrate slide |
JL-FL46 | 果氏巴貝西蟲IgG免疫熒光試劑盒 | Babesia microti IgG IFA Kit |
JL-FL47 | 果氏巴貝西蟲IgM免疫熒光試劑盒 | Babesia microti IgM IFA Kit |
JL-FL48 | 埃立克體IgG微量免疫熒光試劑盒 | Ehrlichia canis Canine IFA IgG Kit |
JL-FL49 | 包柔氏螺旋體菌IgG免疫熒光試劑盒 | Borrelia IgG IFA Kit |
JL-FL50 | 布魯氏菌IgG免疫熒光試劑盒 | Brucella IgG IFA Kit |
JL-FL51 | 里氏新立克次體IgG免疫熒光試劑盒 | Neorickettsia risticii IgG IFA Kit |
JL-FL52 | Toxoplasma IFA Feline IgG Kit | |
JL-FL53 | 弓形蟲IgG免疫熒光試劑盒(檢測狗) | Toxoplasma IFA Canine IgG Kit |
二維碼掃一掃
【公司名稱】 廣州健侖生物科技有限公司
【】 楊永漢
【】
【騰訊 】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-3室
【企業(yè)文化】
與此同時,該研究的結(jié)果也可以應用于人類機體其他組織,比如皮膚、肺部和抗原抗體等;揭示干細胞在人類腸道中的行為機制將是干細胞研究領域的一大飛躍,截至目前研究人員更多的是利用小鼠來進行研究或者是將干細胞置于自然環(huán)境中進行研究,這就扭曲了干細胞的正常行為表現(xiàn);而這項研究中研究者利用開發(fā)的新型工具來揭示腸道干細胞的行為表現(xiàn),這就可以明顯增加研究人員對于腸癌發(fā)生的理解。
zui后,研究者Marnix Jansen博士說道,基于本文的研究,通過對來自腸癌患者的組織進行分析研究,我們就可以清楚揭示腸癌的發(fā)生機理,當然后期還需要進行更為深入的研究才能夠為開發(fā)治療腸癌患者的疾病以及改善其生存質(zhì)量提供幫助。
盡管療法已經(jīng)得到了極大改善,但是兩個成年急性髓性白血病(AML)患者中也僅有一人會生存下去,對于65歲以上的老年人而言,急性髓性白血病平均存活期不到一年;而研究者推測發(fā)病原因可能是白血病干細胞,在患者機體中白血病干細胞并不能*被清除,這些白血病干細胞就是引發(fā)患者病情復發(fā)zui終致死的原因。
近日,刊登在雜志 Cancer Research 上的一篇研究論文中,來自歌德大學( Goethe-Universitat Frankfurt am Main )的研究人員通過研究發(fā)現(xiàn)了急性髓性白血病干細胞的弱點,他們發(fā)現(xiàn)了一種名為5-脂氧合酶(5-LO)的酶類在急性髓性白血病干細胞的生存過程中扮演著重要角色。
At the same time, the results of this study can also be applied to other tissues of human body, such as skin, lungs and antigenic antibodies; to reveal the mechanism of stem cells in human gut will be a big leap forward in the field of stem cell research, as of now researchers More is the use of mice to carry out research or put the stem cells in the natural environment for research, which distorted the normal behavior of stem cells; and researchers in this study using the development of new tools to reveal the intestinal stem cells Behavioral performance, which can significantly increase the researchers understanding of the occurrence of colorectal cancer.
Finally, Dr. Marnix Jansen, a researcher, said that based on the study in this article, we can clearly reveal the mechanism of colorectal cancer by analyzing the tissue from patients with colorectal cancer. Of course, further studies are needed later Development and treatment of diseases of patients with colorectal cancer and improve their quality of life to provide help.
Despite the dramatic improvement in therapies, only one in two adults with acute myeloid leukemia (AML) survives, with an average survival of less than one for acute myeloid leukemia in older adults over 65 years Year; and the researchers speculated that the pathogenesis may be leukemia stem cells, leukemia stem cells in the patient's body can not be compley removed, these leukemia stem cells is caused by the patient's condition eventually lethal cause of death.
Recently, in a research paper published in the International Cancer Research, researchers from Goethe-Universitat Frankfurt am Main discovered the weakness of acute myeloid leukemia stem cells through research that they found in a study entitled 5 - Lipoxygenase (5-LO) enzymes play an important role in the survival of acute myelogenous leukemia stem cells.