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廣州健侖生物科技有限公司

（廣州健侖生物科技有限公司是集研制開發(fā)、銷售、服務于一體的優(yōu)良企業(yè),公司產(chǎn)品涉及臨床快速診斷試劑、食品安全檢測試劑，違禁品快速檢測，動物疾病防疫檢測試劑，免疫診斷試劑、臨床血液學和體液學檢驗試劑、微生物檢驗試劑、分子生物學檢驗試劑、臨床生化試劑、有機試劑等眾多領域，同時核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名診斷產(chǎn)品集團公司產(chǎn)品，致力于為商檢單位、疾病預防控制中心、海關出入境檢疫局、衛(wèi)生防疫單位,緝毒系統(tǒng)，戒毒中心，檢驗檢疫單位、生化企業(yè)、科研院所、醫(yī)療機構(gòu)等機構(gòu)與行業(yè)提供*、高品質(zhì)的產(chǎn)品服務。此外，本公司還開展食品、衛(wèi)生、環(huán)境、藥品等多方面的第三方檢測服務。）

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美國trinity腮腺炎病毒皮肉腫大診斷試劑盒

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【公司名稱】 廣州健侖生物科技有限公司
【市場部】     歐

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【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103室

（1）格子樣變性：格子樣變性與視網(wǎng)膜脫離關系zui為密切。由此產(chǎn)生網(wǎng)膜裂孔者占孔源性脫離眼的40％，約7％的正常眼存在格子樣變性。多見于顳側(cè)或顳上象限的赤道部與鋸齒緣間，呈梭形或條狀，長軸與鋸齒緣平行，病灶內(nèi)視網(wǎng)膜變薄，有許多白色線條，交錯排列成網(wǎng)格狀。這些線條實際上是已經(jīng)閉塞或帶有白鞘的末梢血管。病灶內(nèi)有時可見色素團，來源于視網(wǎng)膜色素上皮。

（2）囊樣變性：好發(fā)于黃斑部及顳下鋸齒緣附近。邊緣清楚，呈圓形或類圓形，暗紅色。周邊部病灶呈網(wǎng)狀，為成簇而略顯高起的小紅點。黃斑囊樣變性呈蜂窩狀。

（3）霜樣變性：大多發(fā)生于赤道部和鋸齒緣附近，網(wǎng)膜表面可見到一些有細小白色或黃色顆粒覆蓋的區(qū)域，厚薄不均，如同覆蓋了一片白霜。此類變性可單獨出現(xiàn)，也可和格子樣變性、囊樣變性同時出現(xiàn)。在赤道部融合成帶狀的稱蝸牛跡樣變性。

（4）鋪路石樣變性：好發(fā)于下方周邊網(wǎng)膜。表現(xiàn)為有色素邊緣的、淡黃色圓形或類圓形、境界清楚的多發(fā)性萎縮病灶，大大小小病灶列成一片呈鋪路石樣。病灶中央部脈絡膜毛細血管萎縮，露出脈絡膜大血管或白色鞏膜。

（5）視網(wǎng)膜加壓發(fā)白與不加壓發(fā)白：將鞏膜壓陷后，眼底的隆起部變?yōu)椴煌该鞯幕野咨Q為加壓發(fā)白。病情進一步發(fā)展時，不加壓也呈灰白色，稱為不加壓發(fā)白，其后緣有時形成一清晰的嵴，多見于上方周邊網(wǎng)膜，被認為是玻璃體牽引的一個指征。

（6）干性視網(wǎng)膜縱向皺襞：皺襞自鋸齒緣的齒緣間向赤道部方向伸展，是過度生長視網(wǎng)膜組織的折疊，皺襞后端受玻璃體牽引易發(fā)生裂孔。玻璃體為一透明狀膠體樣結(jié)構(gòu)，充填于眼球內(nèi)后部4/5的空腔內(nèi)，對視網(wǎng)膜神經(jīng)上皮層貼著于色素上皮層，起著支撐作用。玻璃體變性時，必然發(fā)生玻璃體后脫離，此時玻璃體從視網(wǎng)膜分離，分離往往從眼球的后上部開始，繼而向前擴展。隨著脫離范圍的擴大，甚至僅留下粘連緊密的視盤處。此時眼睛的迅速運動將產(chǎn)生玻璃體的連續(xù)地旋轉(zhuǎn)擺動，使視盤處玻璃體視網(wǎng)膜粘連分離，即形成了玻璃體*脫離。玻璃體脫離時，對其下視網(wǎng)膜有牽拉作用，或引起視網(wǎng)膜裂孔，或使以前已存在的裂孔活化，促進了視網(wǎng)膜脫離的發(fā)生。玻璃體全脫離時，僅在基底部與視網(wǎng)膜粘連，脫離的后部玻璃體以此為支點發(fā)生搖擺，在此處產(chǎn)生明顯的牽拉作用，而周邊網(wǎng)膜又是各種視網(wǎng)膜變性的好發(fā)區(qū)域，非常容易受牽拉造成視網(wǎng)膜裂孔，這也是為什么視網(wǎng)膜脫離時裂孔多位于周邊網(wǎng)膜的原因。

(1) lattice degeneration: lattice degeneration and retinal detachment most closely. The resulting rupture of the omentum accounts for 40% of the holes removed from the eye, and about 7% of the normal eyes have the lattice-like degeneration. More common in the temporal or superior quadrant of the equator between the serrated edge, was spindle or strip, long axis and the serrated edge parallel to the thinning of the lesion within the retina, there are many white lines, staggered arranged in a grid. These lines are actually occluded or white sheathing peripheral blood vessels. Pigmented mass sometimes seen within the lesion, derived from the retinal pigment epithelium.

(2) cystic degeneration: occur in the macula and near the jagged edge of the temporal. Clear edges, round or oval, dark red. Peripheral lesions were reticular, clustered and slightly elevated red dot. Cystoid degeneration was honeycomb.

(3) frost-like degeneration: mostly occurs in the vicinity of the equator and the serrated edge, the surface of the omentum can be seen some small white or yellow particles covered area, uneven thickness, as covered with a white frost. Such degeneration can appear alone, but also and lattice-like degeneration, cystic degeneration appear at the same time. Symptomatic snail-like degeneration that merges into a ribbon at the equator.

(4) stone pavement degeneration: occur in the bottom of the surrounding omentum. Performance for the edge of the pigment, pale yellow round or round, multiple clear atrophy territory, large and small lesions were paved stone-like. Central choroidal capillaries atrophy, showing choroidal blood vessels or white sclera.

(5) retina pressure whitening and no pressure whitening: the sclera after the depression, the raised fundus becomes opaque gray, known as pressure whitening. Further development of the disease, no pressure is gray, known as no pressure whitening, the trailing edge sometimes form a clear crest, more common in the upper peripheral omentum, is considered an indication of vitreous traction.

(6) Longitudinal folds of dry retina: Folds from the edge of the serrated edge to the equatorial extension of the retinal tissue is over-grown folds, folds of the posterior end of the vitreous traction prone to fracture. The vitreous body is a transparent colloid-like structure, which is filled in the cavity of 4/5 in the posterior part of the eyeball, playing a supportive role in attaching the retinal neurocortical layer to the pigment epithelium. Vitreous degeneration, the inevitable vitreous detachment, when the vitreous detachment from the retina, the separation often from the top of the eye after the start, and then move forward. With the extension of the scope of separation, and even leave only the close adhesion of the disc at. At this point the rapid eye movement will produce a continuous rotation of the vitreous swing, the disc at the vitreoretinal adhesions, that is, the formation of a complete detachment of the vitreous. Vitreous detachment, the traction on the lower retina, or cause retinal holes, or to activate the pre-existing hiatus, and promote the occurrence of retinal detachment. Vitreous full detachment, only the basal part of the retina and adhesion, detachment of the posterior vitreous body as a fulcrum swing, where there is a significant pulling effect, while the perineural retina is a variety of retinal degeneration prone areas, Very easy to pull caused by retinal tears, which is why the retinal detachment more than in the surrounding omentum reasons.