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儲(chǔ)存水藏有軍團(tuán)菌病菌檢測(cè)試紙
廣州健侖生物科技有限公司
(廣州健侖生物科技有限公司是集研制開(kāi)發(fā)、銷售、服務(wù)于一體的優(yōu)良企業(yè),公司產(chǎn)品涉及臨床快速診斷試劑、食品安全檢測(cè)試劑,違禁品快速檢測(cè),動(dòng)物疾病防疫檢測(cè)試劑,免疫診斷試劑、臨床血液學(xué)和體液學(xué)檢驗(yàn)試劑、微生物檢驗(yàn)試劑、分子生物學(xué)檢驗(yàn)試劑、臨床生化試劑、有機(jī)試劑等眾多領(lǐng)域,同時(shí)核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名診斷產(chǎn)品集團(tuán)公司產(chǎn)品,致力于為商檢單位、疾病預(yù)防控制中心、海關(guān)出入境檢疫局、衛(wèi)生防疫單位,緝毒系統(tǒng),戒毒中心,檢驗(yàn)檢疫單位、生化企業(yè)、科研院所、醫(yī)療機(jī)構(gòu)等機(jī)構(gòu)與行業(yè)提供*、高品質(zhì)的產(chǎn)品服務(wù)。此外,本公司還開(kāi)展食品、衛(wèi)生、環(huán)境、藥品等多方面的第三方檢測(cè)服務(wù)。)
?常用的免疫膠體金檢測(cè)技術(shù):
(1)免疫膠體金光鏡染色法 細(xì)胞懸液涂片或組織切片,可用膠體金標(biāo)記的抗體進(jìn)行染色,也可在膠體金標(biāo)記的基礎(chǔ)上,以銀顯影液增強(qiáng)標(biāo)記,使被還原的銀原子沉積于已標(biāo)記的金顆粒表面,可明顯增強(qiáng)膠體金標(biāo)記的敏感性。 (2)免疫膠體金電鏡染色法 可用膠體金標(biāo)記的抗體或抗抗體與負(fù)染病毒樣本或組織超薄切片結(jié)合,然后進(jìn)行負(fù)染??捎糜诓《拘螒B(tài)的觀察和病毒檢測(cè)。 斑點(diǎn)免疫金滲濾法 (3)應(yīng)用微孔濾膜(如膜)作載體,先將抗原或抗體點(diǎn)于膜上,封閉后加待檢樣本,洗滌后用膠體金標(biāo)記的抗體檢測(cè)相應(yīng)的抗原或抗體。 (4)膠體金免疫層析法 將特異性的抗原或抗體以條帶狀固定在膜上,膠體金標(biāo)記試劑(抗體或單克隆抗體)吸附在結(jié)合墊上,當(dāng)待檢樣本加到試紙條一端的樣本墊上后,通過(guò)毛細(xì)作用向前移動(dòng),溶解結(jié)合墊上的膠體金標(biāo)記試劑后相互反應(yīng),當(dāng)移動(dòng)至固定的抗原或抗體的區(qū)域時(shí),待檢物與金標(biāo)試劑的結(jié)合物又與之發(fā)生特異性結(jié)合而被截留,聚集在檢測(cè)帶上,可通過(guò)肉眼觀察到顯色結(jié)果。該法現(xiàn)已發(fā)展成為診斷試紙條,使用十分方便。
主要用途:用于檢測(cè)尿樣中嗜肺軍團(tuán)菌血清型1抗原,以支持軍團(tuán)菌感染的診斷。
產(chǎn)品規(guī)格:20T/盒
存儲(chǔ)條件:2-30℃
儲(chǔ)存水藏有軍團(tuán)菌病菌檢測(cè)試紙
我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒:登革熱、瘧疾、西尼羅河、立克次體、無(wú)形體、蜱蟲(chóng)、恙蟲(chóng)、利什曼原蟲(chóng)、RK39、漢坦病毒、深林腦炎、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲(chóng)病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。
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【產(chǎn)品介紹】
貨號(hào) | 產(chǎn)品名稱 | 產(chǎn)品描述 | 產(chǎn)品規(guī)格 | 保存條件 |
JL-ET01 | 免疫捕獲諾如病毒檢測(cè)試劑盒 | 用于檢測(cè)糞便標(biāo)本中的諾如病毒抗原,以支持諾如病毒感染的診斷。 | 20T/盒 | 2-30℃ |
JL-ET02 | 免疫捕獲軍團(tuán)菌檢測(cè)試劑盒 | 用于檢測(cè)尿樣中嗜肺軍團(tuán)菌血清型1抗原,以支持軍團(tuán)菌感染的診斷。 | 20T/盒 | 2-30℃ |
JL-ET03 | 免疫捕獲肺炎鏈球菌檢測(cè)試劑盒 | 用于檢測(cè)尿標(biāo)本中的肺炎鏈球菌抗原,以支持肺炎鏈球菌感染的診斷。 | 20T/盒 | 2-30℃ |
二維碼掃一掃
【公司名稱】 廣州健侖生物科技有限公司
【】 楊永漢
【】
【騰訊 】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-3室
【企業(yè)文化】
填補(bǔ)神經(jīng)元死亡的空間位置,起到修復(fù)和再生作用。神經(jīng)膠質(zhì)細(xì)胞的修復(fù)和再生作用表現(xiàn)為:小膠質(zhì)細(xì)胞可轉(zhuǎn)變?yōu)榫奘杉?xì)胞參與對(duì)損傷組織碎片的清除;膠質(zhì)細(xì)胞特別是星形膠質(zhì)細(xì)胞可通過(guò)增生來(lái)填補(bǔ)缺損,從而起修復(fù)和再生作用。但增生過(guò)強(qiáng)時(shí)則有可能成為引發(fā)腦瘤的病病毒。外周神經(jīng)元軸突的再生也是沿雪旺氏細(xì)胞延伸的。
3.絕緣和屏障作用
神經(jīng)膠質(zhì)細(xì)胞還可起分隔神經(jīng)元的絕緣作用。髓鞘可防止神經(jīng)沖動(dòng)傳導(dǎo)時(shí)的電流擴(kuò)散,對(duì)傳導(dǎo)的絕緣性有重要作用,中樞和外周神經(jīng)纖維的髓鞘分別由少突膠質(zhì)細(xì)胞和雪旺細(xì)胞形成。膠質(zhì)細(xì)胞還參與構(gòu)成血腦屏障,如星形膠質(zhì)細(xì)胞的突起形成的血管周足就是血腦屏障的重要組成部分。膠質(zhì)細(xì)胞的突起有的末端膨大,終止于腦毛細(xì)血管壁上(形成“血管周足”),有的穿行于神經(jīng)元之間,附在神經(jīng)元胞體或樹(shù)突上,可能起血管與神經(jīng)元之間的物質(zhì)運(yùn)輸作用。據(jù)估計(jì),腦毛細(xì)血管表面85%的面積被毛細(xì)血管周足所包繞,構(gòu)成血腦屏障。可選擇性地阻止血液中某些藥物、染料和其他病毒學(xué)物質(zhì)進(jìn)入腦組織。
4.物質(zhì)代謝和營(yíng)養(yǎng)性作用
膠質(zhì)細(xì)胞血液與腦物質(zhì)交換的*途徑,腦內(nèi)各細(xì)胞之間有寬約15~20nm的間隙分離,構(gòu)成容量為腦總量5%的間隙系統(tǒng),內(nèi)充細(xì)胞間液,也是物質(zhì)交換的通道之一。星形膠質(zhì)細(xì)胞的其他突起穿行于神經(jīng)元之間,貼附在胞體和樹(shù)突上,對(duì)神經(jīng)元運(yùn)輸營(yíng)養(yǎng)物質(zhì)和排除代謝產(chǎn)物可能有影響。星形膠質(zhì)細(xì)胞能產(chǎn)生神經(jīng)營(yíng)養(yǎng)病毒子,起支持神經(jīng)元的作用。
5.維持神經(jīng)元的正?;顒?dòng)
神經(jīng)元活動(dòng)時(shí),隨著K+的釋放,細(xì)胞外液中K+濃度將升高,而細(xì)胞外的高K+可能會(huì)干擾神經(jīng)元的正常活動(dòng)。星形膠質(zhì)細(xì)胞可通過(guò)加強(qiáng)膜上病毒-鉀泵的活動(dòng),將細(xì)胞外液中積聚的K+泵入細(xì)胞內(nèi),并通過(guò)細(xì)胞之間的縫隙連接迅速將K+擴(kuò)散到其他神經(jīng)膠質(zhì)細(xì)胞,起到緩沖細(xì)胞外液K+水平的作用,以此限制神經(jīng)元的去極病毒程度,使其興奮性不致過(guò)強(qiáng),有助于神經(jīng)元正?;顒?dòng)的維持。
Fill the spatial location of neuronal death, play a role in repair and regeneration. Repair and regeneration of glial cells showed: microglia can be transformed into macrophages involved in the removal of damaged tissue fragments; glial cells, especially astrocytes can be filled by the proliferation of defects, and thus repair And regeneration. However, hyperplasia is too strong when it may become the brain tumor virus. Regeneration of axons in peripheral neurons also extends along Schwann cells.
Insulation and barrier effect
Glial cells can also play a role in the isolation of neurons. Myelin can prevent the diffusion of current during nerve impulse conduction and plays an important role in conduction insulation. Myelin of central and peripheral nerve fibers is formed by oligodendrocytes and Schwann cells, respectively. Glial cells are also involved in the formation of the blood-brain barrier, such as the formation of astrocytes protruding blood vessels per week is an important part of the blood-brain barrier. Protrusion of glial cells some ends of the enlargement, terminating in the brain capillary wall (the formation of "perivascular"), some through the neurons, attached to the neuronal cell body or dendrites, may play a role in the blood vessels and nerves Meta-material transport between the role. It is estimated that 85% of the area on the surface of the capillaries is surrounded by capillary pericycle, forming the blood-brain barrier. Selectively prevent certain drugs in the blood, dyes and other virological substances into the brain.
4. Material metabolism and nutrition role
The only way for glial cells to exchange blood and brain substance is to separate the cells in the brain by a gap of 15-20 nm in width to form a gap system with a capacity of 5% of the total brain volume. One of the channels. Other astrocytes protrude between neurons and attach to the cell body and dendrites, which may have an impact on neuronal transport of nutrients and exclusion of metabolites. Astrocytes produce neurotrophic virions that act as neurons supporting neurons.
Maintain the normal activities of neurons
With neuronal activity, K + concentration increases in extracellular fluid as K + is released, whereas extracellular extracellular K + may interfere with normal neuronal activity. Astrocytes can pump K + accumulating in the extracellular fluid into the cell by enhancing the activity of the viral-potassium pump on the membrane and rapidly diffuse K + to other glial cells through the gap junction between cells To buffer the extracellular K + level of the role, in order to limit the extent of neuronal depolarization, so that excitability will not be too strong, contribute to the maintenance of normal neuronal activity.